FUJITA Kaori

写真a

Affiliation

Graduate School of Engineering Science  Department of Life Science  Life Science Course 

Research Fields, Keywords

p53, p53 isoforms, cellular senescence, age-related diseases

Graduating School 【 display / non-display

  • 1991.04
    -
    1995.03

    Tohoku University   Faculty of Pharmaceutical Science   Graduated

Graduate School 【 display / non-display

  • 1997.04
    -
    2000.03

    Tohoku University  Graduate School, Division of Pharmaceutical Sciences  Doctor's Degree Program  Completed

  • 1995.04
    -
    1997.03

    Tohoku University  Graduate School, Division of Pharmaceutical Sciences  Master's Degree Program  Completed

Campus Career 【 display / non-display

  • 2021.04
    -
    Now

    Akita University   Graduate School of Engineering Science   Department of Life Science   Life Science Course   Lecturer  

External Career 【 display / non-display

  • 2010.01
    -
    2012.01

    National Cancer Institute, National Institutes of Health, USA   Laboratory of Human Carcinogenesis   Federal Employee, Research Fellow  

  • 2005.02
    -
    2009.01

    National Cancer Institute, National Institutes of Health, USA   Laboratory of Human Carcinogenesis   Visiting Fellow  

Academic Society Affiliations 【 display / non-display

  • 2019.12
    -
    Now
     

    UNITED KINGDOM

     

    International Cell Senescence Association (ICSA)

Research Field (grants-in-aid-for-scientific-research classification) 【 display / non-display

  • General medical chemistry

  • Cell biology

  • Pathological medical chemistry

  • Orthopaedic surgery

Qualification acquired 【 display / non-display

  • Pharmacist

 

Published Papers 【 display / non-display

  • p53 isoforms in cellular senescence- and ageing-associated biological and physiological functions.

    Kaori FUJITA

    International Journal of Molecular Sciences   20 ( 23 ) 6023   2019.11  [Refereed]  [Invited]

    Single author

  • Positive feedback between p53 and TRF2 during telomere-damage signaling and cellular senescence.

    Kaori FUJITA, Izumi Horikawa, Abdul M Mondal, Lisa M Miller Jenkins, et al.

    Nature Cell Biology   12 ( 12 ) 1205 - 1212   2010.12  [Refereed]

    International Co-author

    DOI

  • p53 isoforms Δ133p53 and p53β are endogenous regulators of replicative cellular senescence.

    Kaori FUJITA, Abudal M Mondal, Izumi Horikawa, Giang H Nguyễn, et al.

    Nature Cell Biology   11 ( 9 ) 1135 - 1142   2009.09  [Refereed]

    International Co-author

    DOI

  • Autophagic degradation of the inhibitory p53 isoform Δ133p53α as a regulatory mechanism for p53-mediated senescence.

    Izumi Horikawa, Kaori FUJITA, Lisa M Miller Jenkins, Yukiharu Hiyoshi, et al.

    Nature Communications   5   4706   2014.08  [Refereed]

    International Co-author

    DOI

  • p53 isoforms regulate aging- and tumor-associated replicative senescence in T lymphocytes.

    Abdul M Mondal, Izumi Horikawa, Sharon R Pine, Kaori FUJITA, et al.

    International Journal of Clinical Investigation   123 ( 12 ) 5247 - 5257   2013.12  [Refereed]

    International Co-author

    DOI

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Grant-in-Aid for Scientific Research 【 display / non-display

  • Grant-in-Aid for Scientific Research(B)

    Project Year: 2018.04  -  Now 

  • Grant-in-Aid for Challenging Research (Pioneering)/(Exploratory)

    Project Year: 2014.04  -  2016.03 

  • Grant-in-Aid for Scientific Research(B)

    Project Year: 2013.04  -  2017.03